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Journal Article 1: Beaver, K. M., Eagle Schutt, J., Boutwell, B. B., Ratchford, M., Roberts, K., & Barnes, J. C. (2009). Genetic and environmental influences on levels of self-control and delinquent peer affiliation: Results from a longitudinal sample of adolescent twins. Criminal Justice and Behavior, 36, 41–60.
Abstract: Despite the fact that low self-control and exposure to delinquent peers are two of the most robust and consistent predictors of crime, delinquency, and antisocial behavior, much remains unknown about what causes self-control to develop and what causes youths to befriend antisocial peers. This study estimated the relative effects of environmental and genetic factors on levels of self-control and contact with delinquent peers in a sample of twins from the National Longitudinal Study of Adolescent Health (Add Health). DeFries-Fulker analysis of the Add Health data revealed that both self-control and contact with drug-using friends were influenced by genetic factors and the nonshared environment, whereas the shared environment exhibited relatively small and inconsistent effects. Implications for self-control theory and social learning theory are discussed.

Journal Article 2: Martens, W. H. (2002). Criminality and moral dysfunctions: Neurological, biochemical, and genetic dimensions. International Journal of Offender Therapy and Comparative Criminology, 46, 170–182.
Abstract: In this article, the author attempts to demonstrate a relationship between neurobiological dysfunctions and/or genetically determined deviant behavior and personality traits as well as moral abnormalities. Data from neuroscience show that a number of neurological dysfunctions are linked to cognitive and emotional disturbances. Cognitive and emotional abnormalities, in turn, are frequently related to moral dysfunctions. Moreover, neurological disorders can produce dramatic psychological and social problems, personality changes, and behavioral problems in patients. Those mental, emotional, and psychosocial problems and related moral dysfunctions are frequently linked to violence and/or criminal behavior. Genetic research found evidence of inheritability of antisocial traits, which interfere with moral development and activities. This information has consequences for any assessment and disposition within the legal system. More research on the interrelationship between neuro(bio)logical, genetic, emotional, and mental aspects of moral dysfunctions is needed for the development of adequate treatment, prevention, and intervention programs.

Journal Article 3: Mysterud, I., & Poleszynski, D. V. (2003). Expanding evolutionary psychology: toward a better understanding of violence and aggression. Biology and Social Life, 42, 5–50.
Abstract: The “mainstream” evolutionary psychology model is currently under criticism from scientists of other persuasions wanting to expand the model or to make it more realistic in various ways. We argue that focusing on the environment as if it consisted only of social (or sociocultural) factors gives too limited a perspective if evolutionary approaches are to understand the behavior of modern humans. Taking the case of violence, we argue that numerous novel environmental factors of nutritional and physical-chemical origin should be considered as relevant proximate factors. The common thesis presented here is that several aspects of the biotic or abiotic environment are able to change brain chemistry, thus predisposing individuals to violence and aggression in given contexts. In the past, aggressive behavior has had a number of useful functions that were of particular importance to our ancestors' survival and reproduction. However, some of the conditions in our novel environment, which either lowered the threshold for aggression or released such behavior in contexts which were adaptive in our evolutionary past, no longer apply. It is high time evolutionary approaches to violence are expanded to include the possibilities that violence may be triggered by nutritionally depleted foods, reactive hypoglycemia caused by habitual intake of foods with a high glycemic index (GI), food allergies/intolerances and exposure to new environmental toxins (heavy metals, synthetic poisons).